Hematoxylin-eosin (HE) staining showed the accumulation of leucocytes in the intestinal mucosa. Intestinal motility of mice was inhibited, and intestinal permeability was increased after stroke. Ischemic stroke was made as a disease model in mice, in which brain and ileal tissues were collected for experiments on the 1 st and 7 th day after stroke. The current study is to investigate the changes of intestinal structure and function in mice after ischemic stroke. However, there are few studies focusing on the GI tract after stroke. Patients who have a stroke are susceptible to many gastrointestinal (GI) complications, such as dysphagia, GI bleeding, and fecal incontinence. Stroke is the second leading cause of death worldwide.
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